Text book of comparative general pathology for practitioners and students of veterinary medicine / by Th. Kitt; authorized translation by William W. Cadbury ; edited with notes and additional illustrations by Allen J. Smith.

  • Kitt, Th. (Theodor), 1858-1941.
Date:
1906
Catalogue details

Licence: In copyright

Credit: Text book of comparative general pathology for practitioners and students of veterinary medicine / by Th. Kitt; authorized translation by William W. Cadbury ; edited with notes and additional illustrations by Allen J. Smith. Source: Wellcome Collection.

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    iag or induration of the liver), occupation of spaces and formation of false membranes by fibrinous material (pleurisy, croup), micro- scopic changes in the cells (karyolysis, fatty infiltration). (2) Chemico-pathological processes: Quantitative and qualitative changes of the chemical gonstituents of the animal organism and its parts (metabolic faults), as the presence of bile in the blood, albu- men in the urine, uric acid in the joints or abnormal proportion of water in the blood. [Here might well be included, too, the ex- cessive formation or uric acid or its salts, of the faulty carbohy- drate changes of diabetes, the development of faulty types of albumen in the blood and within the cells, the toxic changes of urremia and other intoxications, those essentially chemical processes which underlie in an important degree the changes produced by infections, as well as a wide group of more or less indefinite meta- bolic diseases.] (3) Functional-pathological or symptomatic disturbances (some- times spoken of as dynamic disturbances) : These are faults in the nicety of balance of the various physiological activities of the or- ganism, as the occurrence of convulsions, pain, unconsciousness, labored breathing, diarrhcea or dribbling urine. Functional dis- turbances are usually. caused by structural and metabolic faults. Commonly the latter are primary, as in physiological life the effi- ciency, growth and development of the organs are dependent upon the chemical processes of nutrition and metabolism. Our knowl- edge of the morphological and chemical basis of functional de- rangements is; however, incomplete. Consideration, description and investigation of pathological variations may therefore bedivided into (a) Pathological Morpliology or Anatomy, (b) Pathological Chemistry and (c) Symptomatology, together with (d) considerations of the influences under which dis- eases develop, and the actual causes of disease (Aetiology: aWla, cause). In the origin of disease some cause is apt to act so as to bring about material changes, chemical and physical, pathological lesions (Iccdere, to injure) of the coniponent elements and organs, as well as the counteraction or pathological reaction on the part of these same elements and organs, both of which manifest them- selves by functional disturbances (symptoms or signs of disease). That phase of pathology which seeks to explain the development of lesions and symptoms constitutes Pathogenesis (»} 7hcais, de- velopment, beginning) ; that which concerns the observation of the chronological succession of disease-events, the onset, course