An index of diseases and their treatment / by Thomas Hawkes Tanner.

  • Thomas Hawkes Tanner
Date:
1883
Catalogue details

Licence: Public Domain Mark

Credit: An index of diseases and their treatment / by Thomas Hawkes Tanner. Source: Wellcome Collection.

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    to real vec;clable starcli, being round or oval, presenting concentric layers—f!^. the little corpora amylacea of nervous syslem ; laminalod bodies found in prostate of every adult man, and_ wliicli fionietinics iurm prostatic concretions; and rare forms of a similar kind winch occur in certain conditions of lungs. (2) In foregoing cases the starch-like matter lies hetireen the ele- ments of the tissu'es. Very dilfcrent are those cases where there is a de-eneration of the tissues themselves,—the deposited material here albuminoid and not starchy. The change begins in the capillary walls and in muscular fibre-cells of small arteries: walls of vessels get o-radually thickened, while their calibre diminishes. Tlicn the morbid process'involves surrounding anajmic parenchyma; extending until whole tissue in neighbourhood of arteries is altered. _ Several organs are cjenera lly invad'ed simultaneously, and rendered incapable of per- forming- their functions. Patients gradually assume a cachectic, broken-down appearance; lose He.sh and strength; dropsy often super- venes ; urine gets albuminous if kidneys be affected; diarrhcea sets in when digestive tract is involved ; and in spite of remedies dcatli soon takes place. ^V\len liver, spleen, or kidneys arc organs afleeted, an unpractised eye may fail to detect alteration in structure unless there be an ex- treme amount of disease, when they arc large, firm, heavy, and on section glistening. When a liver is incised where amyloid degenera- tion is far advanced, a feeling is communicated like that expei ieiiced on passing a knife through wax: cut surface presents a semi-trans- parent appearance. The gland is increased in size ; has some resem- blance to a fatty liver, but its greater weight and firmness on handling distinguish it. If disease be very extensive, no trace cf normal structure can bo distinguished, though in an earlier stage the lobules are seen distinctly mapped out, owing to the matter being deposited within the lobule and in and among secreting cells. Amyloid degeneration rarely exists alone, but is most commonly met with as a secondary consequence of prolonged supiuiration, tuber- culosis, disease of bones, and syphilis. Thus, in phthisis, this form of hepatic disease is probably more common than fatty liver ; while sometimes amyloid and fatty degeneration occur togctlier._ So fre- quently has amyloid degeneration been found connected witli caries or necrosis, that it was thought the osseous disease exercised soino determining infiuence on its' production, but amyloid degeneration is as frequently associated with plithisis and syphilis as with bone_ dis- ease. So it was considered that Bright's disease was often associated with amyloid degeneration, until the discovery that the former was sometimes merely a symptinn of the latter afiecting kidneys. It lias been supposed to be caused by the abstraction of potash salts from the blood.—Sec Jfqxdic Degenerations; Jlenal Vc//ciirra'!oiii^, &e. ANASARCA.—From 'Avd, through ; adp^, the (lesli. Syiion. JlijilroiiH (rlhihiris ToliitH Corporis; JI ijil rnxurca ; General Projis//.^ —The more or less general accumulatiun of fccruin in the meshes ol
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