Catalogue of the museum of the Royal London Ophthalmic Hospital, Moorfields : part II inflammations.

  • Collins, E. Treacher (Edward Treacher), 1862-1937
Date:
1894
Catalogue details

Licence: Public Domain Mark

Credit: Catalogue of the museum of the Royal London Ophthalmic Hospital, Moorfields : part II inflammations. Source: Wellcome Collection.

Provider: This material has been provided by The Royal College of Surgeons of England. The original may be consulted at The Royal College of Surgeons of England.

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    anteiior capsule, wliicli is brought about by the contact of the lens and the inflamed cornea. In some cases the opacities are raised above the surface of the rest of the lens in a pyramidal form (No. 20; Fig. 8), in others they are flat (Nos. 22, 24, 26, and 30). Tags of membrane at times pass between the scar tissue in the cornea and the centre of the capsule of the lens (Nos. 20 and 28). When the lens and vitreous have escaped through a perforating ulcer of the cornea, the eye collapses and be- comes shrunken and puckered. So great is the change that some of these shrunken eyes undergo, that tlie in- dividual parts of them cannot be clearly differentiated microscopically. In most of them the sclerotic, though much diminished in area, is considerably increased in thickness (Nos. 16, 18, and 37). It also becomes puckered, the folds which form in it being usually situated beneath the insertion of each rectus muscle. In eyes in which the cornea has become ulcerated in the course of an ophthalmia, the inflammation is frequently not limited to it. The uveal tract becomes involved, and inflammatory effusions are poured out into the anterior chamber (No. 3) and ^dtreou8. Frequently the latter becomes shrunken and detached (Nos. 5, 15, 22, and 27), ' sometimes the retina is detached also (No. 15). As the result of inflammation or stretching the choroid is fre- quently found atrophied (Nos. 13, 15, 22, 27, 31, 36, and 39). Occasionally bone develops on its inner surface (No. 23). The pigment epithelial layer commonly under- goes extensive changes, the cells composing it growing into the substance of the retina and giving rise to patches of various irregular shapes and sizes (Nos. 11, 13, 21, 27, and 36). No. 1.—The outer half of the left e^-e of a girl, aged 2| years. When she was six months old its cornea was ulcerated. She was very ill at that time and not expected to live, though without any deflnite malady. There was never much discharge from the tyes. Theie is a large, thick, stajAiylomatous cornea.
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