Visceral angioneuroses : (the visceral crises of vasomotor ataxia) / by Solomon Solis Cohen.

  • Solomon Solis-Cohen
Date:
1910
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Licence: In copyright

Credit: Visceral angioneuroses : (the visceral crises of vasomotor ataxia) / by Solomon Solis Cohen. Source: Wellcome Collection.

Provider: This material has been provided by The Royal College of Surgeons of England. The original may be consulted at The Royal College of Surgeons of England.

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    vestigation,- however, such care may be taken for granted. it is somewhat difficult to estimate the importance of the history, given by adults, of eruptive fevers in childhood; especially when “third and “fourth” at- tacks are spoken of. I have seen, both in children and in* adults, angioneurotic storms mistaken for measles, roseola, or scarlet fever. I have myself made the mistake of quarantining for scarlet fever a boy whose family history, previous personal his- tory, and physical characteristics would nowadays put me on guard. The attack was one of erythema, with repeated desquamation. In a case observed at Jefferson Hospital, erythema and angioneurotic oedema developed while the patient was being ex- hibited to the class at a clinical lecture, and the parents volunteered the statement that the appear- ance was the same as what they had supposed, in a previous attack, to be scarlet fever. In the case of a boy seen at the Polyclinic Hospital, on removing the clothing for examination there appeared first a general erythema, soon replaced by rude geo- metrical designs in red, white, and blue lines and patches. This is the patient I have referred to else- where as the'“tatooed boy.” In the case of pseudo- angina previously cited, similar exposure produced red and white circular patches. I have seen many curious forms of transient rashes—red; red and white ; red, white, and blue; red and blue; but never blue and white. The scarlatiniform and rubelloid attacks are fre- quently accompanied with nausea, vomiting, abdom- inal pain, sometimes albuminuria, not rarely fever; and are probably of toxic excitation. As isolated occurrences, adequately caused, they would be of no importance other than that derived from their aeti- ology. When inadequately caused, or forming part
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