Report on the progress of pathology, practical medicine, and therapeutics, for the years 1842-3-4 / by James Risdon Bennett.

  • James Risdon Bennett
Date:
1845
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Licence: Public Domain Mark

Credit: Report on the progress of pathology, practical medicine, and therapeutics, for the years 1842-3-4 / by James Risdon Bennett. Source: Wellcome Collection.

Provider: This material has been provided by The Royal College of Surgeons of England. The original may be consulted at The Royal College of Surgeons of England.

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    In a second class of cases similar results ensue from defective expansion of theluno-s arising from mcclianical pressure exerted on them or the air-passaps, e.g. in deformity of the spine or chest,orpleurisy and consequent adhesions; unless the opposite lung take on a compensating action, when, in consequence of its increased activity, there is great danger of tubercular deposition. His tinrd class includes a set of cases the true nature ot which he thinks has hitherto escaped the notice of pathologists, in which the obstruction to the circulation on the rio-ht side of the heart is the result of pericarditis acting mediately througli the impediments which it offers to the respiratory movements. The state ot respi- ration so characteristic of pericarditis cannot, he argues, continue long in a person whose growth is not yet completed, without otfering great obstruction to the development of the lungs,and thusinducing important changes in the righ side of the heart, and the evils thence resaking. Theconsequences of pericarditis are thus very different in the adult, from those ensuing in a person whose frame is not yet fully developed. Dr. Barlow denies that hypertrophy ol the heart is a necessary consequence of pericardial adhesion, and adduces illustrative of this, in which a complete ring of ossific matter (deposited in the false membrane, forming the medium of adhesion between the two surfaces of the pericardium), surrounded the base of the heart. I he man had been sub- ject of rheumatic pericarditis two years before, '^he heart itself was than natural. The true cause of hypertrophy and dilatation, with Fn^rd'^ adhesions, is the impediment to the circulation through the lungs, and not im- peded action of the heart from its being shackled by In connexion with this subject, he also alludes to the arrest of the heart s and inability to carry on the circulation from this cause, as quence of pericarditis in young people, and cites a remarkable case of atrophy ‘^^nthe fmirth set of cases the defective expansion of quence of obstruction in the left heart in Contagious cells. Inoculation hy means . . . heqUi,y die gesammte Med.’ states that he has succeeded m aniiTuils—carcinoma, tubercle, melanosis, condy omata, warts, ozeena, and coryza-charbon (malignant pustule), and hydrophobia—^ the cells of these several diseases, and mentions as important practical facts, tl^^^^ Hircells of recent coryza are readily destroyed by the action ofchloride o m but if the disease becomes chronic, the cells disappear and dan°-erous confervse of ozoena. The cells of charbon are so contagious that it is dan»ero s to inoculate with them ; after having subjected them to P them in lime for fifteen days, he was able to inoculate a small goat 1 h® Gooch, Mayo, and Langenbeck.] nn elaborate and valuable paper Phthisis, Jnjluence of employment on. of phthisis, Uie varies inversely with the amount ot ° jre to a liicrli temperature, nor * Prov. Med. and Surg. Journal, No. 15C. Journal of the Statistical Society.