An introduction to pathology and morbid anatomy / by T. Henry Green ; revised and enlarged by H. Montague Murray.

  • Green, T. Henry (Thomas Henry), 1841-1923
Date:
1900
Catalogue details

Licence: In copyright

Credit: An introduction to pathology and morbid anatomy / by T. Henry Green ; revised and enlarged by H. Montague Murray. Source: Wellcome Collection.

Provider: This material has been provided by King’s College London. The original may be consulted at King’s College London.

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    2. By the carnage of the caiuses of disease from a primary focm lo parts at a distance.—Thus organisms may be carried by the lymphalicx, and give rise to inflamed lymphatic glands; pieces of clot may be conveyed by the blood-vessels, and produce embolism; and a renal calculus may be transferred through the ureter to the bladder. 3. iH«'A«?;(6Y///_j/, by so-called hack-lelling.—Thus stricture of the urethra causes hypertrophy of the bladder, if the obstacle to the outflow of iuine can thus be overcome; or simple dilatation of the bladder,if its efforts are futile. In either case, the difficulty of entry of urine into the bladder is increased, and the uretei-s, pelves, and kidneys dilate. Interstitial nephritis results from the pressure, the renal functions are imperfectly pei'formed, and this is detrimental to the organism at large. The succession of changes which result from incompetence of the mitral valves is another familiar example of this mode of extension of disease. (See Passive HyperEemia.) 4. Failure of any pari to do its share of work in the economy.—The result of such failure will depend upon the readiness and completeness with which its defects can be compensated. If the Avork can be readily taken over by other parts, as can that of a sweat or sebaceous gland, nothing is noticed ; on the other hand, extirpation of a kidney which was doing work, is followed by a time of danger from the consequent interference with the excretion and intei'nal secretion of renal products, as the other kidney is at first unequal to the double duty. Absolute failure of the cardiac or of the respu'atory function will cause death, there being no power of compensation. TERMINATIONS OF DISEASE The possible termina- tions of disease are recoveiy, or return of the part to the discharge of its normal functions ; partial recovery ; and death, or com^jlete cessation of function. Certain diseases can scarcely be said to have a termina- tion ; when once established they remain stationary. It will be useful here to give a list of the morbid processes to which all organs are more or less liable :— Developmental errors. Necrosis. * * * Inflammation. Results of injury. Regeneration. Displacement. Hypertrophy. Haemorrhage. * » Tumour-formation. * * * ' Anaemia. Hyperaemia. Lodgment of parasites. QJdema. „ . ... . , Stricture and its consequences may 'P occur in jmy duct or canal; and cal- Degeneration. p„]j ,j^.jy (]evelop in any of them.
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